Partial lesion of thalamic ventral intermediate nucleus after chronic high‐frequency stimulation
Identifieur interne : 00A892 ( Main/Exploration ); précédent : 00A891; suivant : 00A893Partial lesion of thalamic ventral intermediate nucleus after chronic high‐frequency stimulation
Auteurs : Jasmine Henderson [Australie] ; Michael Rodriguez [Australie] ; Dudley O'Sullivan [Australie] ; Malcolm Pell [Australie] ; Victor Fung [Australie] ; Alim Louis Benabid [Australie, France] ; Glenda Halliday [Australie]Source :
- Movement Disorders [ 0885-3185 ] ; 2004-06.
Descripteurs français
- Pascal (Inist)
- Wicri :
- topic : Neurologie.
English descriptors
- KwdEn :
- Benabid, Brain stimulation, Cannabis, Cell loss, Chronic stimulation, Deep brain stimulation, Disord, Electrode, Electrode track, Electrode tract, Gliosis, Halliday, High frequency, Lewy, Lewy bodies, Mild gliosis, Movement disorders, Myoclonus, Nervous system diseases, Neurol, Neurology, Neuron, Neuronal, Neuronal loss, Propriospinal, Propriospinal myoclonus, Pulse width, Spinal, Substantia nigra, Thalamic, Thalamic nucleus, Thalamic stimulation, Tremor, Tremor improvement, Unmedicated state, Unoperated side, Upper limb, Ventral, Ventral nucleus, Years postoperatively.
- Teeft :
- Benabid, Brain stimulation, Cannabis, Cell loss, Chronic stimulation, Deep brain stimulation, Disord, Electrode, Electrode track, Electrode tract, Gliosis, Halliday, Lewy, Lewy bodies, Mild gliosis, Movement disorders, Myoclonus, Neurol, Neurology, Neuron, Neuronal, Neuronal loss, Propriospinal, Propriospinal myoclonus, Pulse width, Spinal, Substantia nigra, Thalamic, Thalamic nucleus, Thalamic stimulation, Tremor, Tremor improvement, Unmedicated state, Unoperated side, Upper limb, Ventral, Years postoperatively.
Abstract
A 73‐year‐old man with Parkinson's disease underwent thalamic stimulation for disabling tremor with excellent results only when stimulation on. Post‐mortem neuropathology (7 years postoperatively) revealed 60% cell loss within 0.5 mm of the electrode tip. Tremor improvement was attributable to chronic stimulation, not microthalamotomy. © 2004 Movement Disorder Society
Url:
DOI: 10.1002/mds.10709
Affiliations:
- Australie, France
- Auvergne-Rhône-Alpes, Nouvelle-Galles du Sud, Rhône-Alpes
- Grenoble, Sydney
- Université de Sydney
Links toward previous steps (curation, corpus...)
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Le document en format XML
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<term>Chronic stimulation</term>
<term>Deep brain stimulation</term>
<term>Disord</term>
<term>Electrode</term>
<term>Electrode track</term>
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<term>Gliosis</term>
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<term>High frequency</term>
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<term>Mild gliosis</term>
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<term>Nervous system diseases</term>
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<term>Neurology</term>
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<term>Propriospinal</term>
<term>Propriospinal myoclonus</term>
<term>Pulse width</term>
<term>Spinal</term>
<term>Substantia nigra</term>
<term>Thalamic</term>
<term>Thalamic nucleus</term>
<term>Thalamic stimulation</term>
<term>Tremor</term>
<term>Tremor improvement</term>
<term>Unmedicated state</term>
<term>Unoperated side</term>
<term>Upper limb</term>
<term>Ventral</term>
<term>Ventral nucleus</term>
<term>Years postoperatively</term>
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<front><div type="abstract" xml:lang="en">A 73‐year‐old man with Parkinson's disease underwent thalamic stimulation for disabling tremor with excellent results only when stimulation on. Post‐mortem neuropathology (7 years postoperatively) revealed 60% cell loss within 0.5 mm of the electrode tip. Tremor improvement was attributable to chronic stimulation, not microthalamotomy. © 2004 Movement Disorder Society</div>
</front>
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